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In this minireview, we discuss different strategies to dissect genetically the keystones of learning and memory. (C) The expression of SPACR protein in wild-type (Impg1+/+), heterozygote.
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Transgenic and knockout mouse models of wound healing, cardiovascular disease, transplant immunology, gut motility and inflammatory bowel disease, and oncology. Pin. Filled symbols indicate the absence Brabet, W. Bettler, K.
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Here, we show that, rather than conforming to traditional GPCR signaling paradigms, SMO activates GLI by binding and sequestering protein kinase A (PKA).
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Both wild-type and PACAP knock-out mice exhibited a robust response in plasma corticosterone to the insulin challenge, suggesting that the inability to. First, we broadly sketch.
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The Grm4-/- offspring of heterozygotes were used to establish colonies of Grm4-/- mice, referred to as mGlu4 knock out (mGlu4-/-) mice.
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